Advances in Parasitology, Vol. 17 by W.H.R. Lumsden, R. Muller, J.R. Baker (Eds.)

By W.H.R. Lumsden, R. Muller, J.R. Baker (Eds.)

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Nascimento, 0. , Martins Campos, J. , Quarentii, G. and Marcondes, E. ( 1 970). Giardiasis in childhood. Absorption tests and biochemistry, histochemistry, light and electron microscopy of jejunal mucosa. Archives of Disease in Childhood 45, 466472. Barbour, A. , Nichols, C. R. and Fukushima, T. (1976). An outbreak of giardiasis in a group of campers. American Journal of Tropical Medicine and Hygiene 25, 384389. Barnes, G. L. and Kay, R. (1977). Blood groups in giardiasis. Lancet i, 808. , Farid, Z .

1974). Morphologic alterations in the microvillous border of villous epithelial cells produced by intestinal microorganisms. American Journal of Clinical Nutrition 27, 1277-1286. Eyles, D. , Jones, F. E. and Smith, C. S. (1953). A study of Entamoeba histolytica and other intestinal parasites in a rural West Tennessee community. American Journal of Hygiene 2, 173-190. Fantham, H. B. and Porter, A. (1916). The pathogenicity of Giardia (Lamblia) intestinalis to men and to experimental animals. British Medical Journal 2, 139-141.

1964) postulate that the lesions and symptoms that occur in giardiasis may result from intestinal bacteria or fungi in association with the protozoa, although bacteria or fungi were not seen in or near the lesions in the cases they presented. LeonBarua (1968) subscribed to this hypothesis and presented as evidence the fact that tetracyclines sometimes suppress the patient’s diarrhoea while the Giurdia infection is still present. Tomkins et al. (1976, 1978) made qualitative and quantitative studies of the small intestinal bacteria present in Giurdia infections; their work is the strongest evidence to date that bacteria may potentiate the mucosal lesions that occur in giardiasis and may be responsible for symptomatic malabsorption.

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